{"id":57585,"date":"2025-08-26T22:26:09","date_gmt":"2025-08-26T16:56:09","guid":{"rendered":"https:\/\/e-gmat.com\/blogs\/?p=57585"},"modified":"2025-09-05T00:21:33","modified_gmt":"2025-09-04T18:51:33","slug":"in-a-study-researchers-repeatedly-measured-the-thickness-of","status":"publish","type":"post","link":"https:\/\/e-gmat.com\/blogs\/in-a-study-researchers-repeatedly-measured-the-thickness-of\/","title":{"rendered":"In a study, researchers repeatedly measured the thickness of a specific artery in each….."},"content":{"rendered":"A <\/span> 4<\/span> min read <\/span><\/span>\n

In a study, researchers repeatedly measured the thickness of a specific artery in each of thousands of volunteers over several years. The researchers found during the study that the artery became thicker more quickly in individuals who lived in cities with significant air pollution. Since thick arterial walls are associated with heart disease, the researchers concluded that exposure to significant urban air pollution contributes to heart disease.<\/p>\n\n\n\n

In order to assess the force of the researchers’ evidence for their conclusion, it would be most helpful to know whether<\/p>\n\n\n\n

A. any of the volunteers whose arteries became thicker during the study lived in areas without urban air pollution<\/p>\n\n\n\n

B. the specific artery the researchers examined is fairly typical of arteries in the human body with respect to the thickness of its walls<\/p>\n\n\n\n

C. any factors that are more common in urban areas contribute to heart disease without contributing to the thickening of arterial walls<\/p>\n\n\n\n

D. any of the volunteers whose arteries were among the thickest at the end of the study had arteries that were among the thickest when the study began<\/p>\n\n\n\n

E. any other environmental factors tend, in cities with significant amounts of air pollution, to act to thicken arterial walls more quickly than they do in cities with lower amounts of air pollution<\/p>\n\n\n\n

Solution<\/h2>\n\n\n\n

Passage Analysis:<\/h3>\n\n\n\n
Text from Passage<\/strong><\/td>Analysis<\/strong><\/td><\/tr>
In a study, researchers repeatedly measured the thickness of a specific artery in each of thousands of volunteers over several years.<\/em><\/td>What it says:<\/strong> Researchers tracked artery thickness in thousands of people over multiple years
What it does:<\/strong> Sets up the study design and shows we have long-term data on many people
What it is:<\/strong> Study methodology description
Visualization:<\/strong> Thousands of volunteers \u2192 ~5,000 people, measured over several years \u2192 ~3-5 years of data collection<\/td><\/tr>
The researchers found during the study that the artery became thicker more quickly in individuals who lived in cities with significant air pollution.<\/em><\/td>What it says:<\/strong> People in polluted cities had faster artery thickening than others
What it does:<\/strong> Presents the key finding that connects air pollution to artery changes
What it is:<\/strong> Study finding\/evidence
Visualization:<\/strong> Significant pollution cities \u2192 ~40-50% higher pollution levels, artery thickening rate \u2192 polluted city residents: 2x faster thickening vs. clean air residents<\/td><\/tr>
Since thick arterial walls are associated with heart disease, the researchers concluded that exposure to significant urban air pollution contributes to heart disease.<\/em><\/td>What it says:<\/strong> Thick arteries link to heart disease, so researchers concluded pollution causes heart disease
What it does:<\/strong> Connects the artery findings to a broader health conclusion using existing medical knowledge
What it is:<\/strong> Author’s conclusion based on study + background medical fact
Visualization:<\/strong> Thick arterial walls \u2192 ~30-40% increased heart disease risk, pollution exposure \u2192 contributes to heart disease development<\/td><\/tr><\/tbody><\/table><\/figure>\n\n\n\n

Argument Flow:<\/h3>\n\n\n\n

The argument starts with describing a long-term study design, then presents the key finding that pollution correlates with faster artery thickening, and finally uses medical knowledge about thick arteries to conclude that pollution contributes to heart disease.<\/p>\n\n\n\n

Main Conclusion:<\/h3>\n\n\n\n

Exposure to significant urban air pollution contributes to heart disease.<\/p>\n\n\n\n

Logical Structure:<\/h3>\n\n\n\n

The researchers use a two-step logical chain: (1) their study shows pollution leads to thicker arteries, and (2) existing medical knowledge shows thick arteries are associated with heart disease, therefore (3) pollution contributes to heart disease. This creates a cause-and-effect argument linking pollution \u2192 thick arteries \u2192 heart disease.<\/p>\n\n\n\n

Prethinking:<\/h3>\n\n\n\n

Question type:<\/h4>\n\n\n\n

Evaluate – We need to find what additional information would help us assess how strong or weak the researchers’ evidence is for their conclusion that air pollution contributes to heart disease<\/p>\n\n\n\n

Precision of Claims<\/h4>\n\n\n\n

The study found a correlation between living in polluted cities and faster artery thickening. The conclusion makes a causal claim that pollution contributes to heart disease based on this correlation plus the fact that thick arteries are associated with heart disease<\/p>\n\n\n\n

Strategy<\/h4>\n\n\n\n

For evaluate questions, we need to think of key assumptions the argument makes, then create scenarios that would either strengthen or weaken the conclusion when we know whether they’re true or false. The main logical gap here is jumping from correlation to causation – just because people in polluted cities had thicker arteries doesn’t necessarily mean pollution caused it. We should think about alternative explanations and what information would help us rule them in or out<\/p>\n\n\n\n

Answer Choices Explained<\/h3>\n\n\n\n

A. any of the volunteers whose arteries became thicker during the study lived in areas without urban air pollution<\/p>\n\n\n\n

This asks whether any volunteers with thicker arteries lived in non-polluted areas. While this might provide some comparative information, it doesn’t directly help us assess whether pollution is actually causing the artery thickening in polluted cities. Even if some people in clean areas also had thick arteries, that wouldn’t tell us much about the pollution-artery relationship since there could be many other causes of artery thickening.<\/p>\n\n\n\n

B. the specific artery the researchers examined is fairly typical of arteries in the human body with respect to the thickness of its walls<\/p>\n\n\n\n

This asks whether the specific artery studied is typical of arteries in general. This is about the generalizability of the findings to other arteries, but it doesn’t help us evaluate whether the evidence actually supports the causal claim about pollution causing heart disease. The strength of the causal argument remains the same regardless of whether this artery is typical.<\/p>\n\n\n\n

C. any factors that are more common in urban areas contribute to heart disease without contributing to the thickening of arterial walls<\/p>\n\n\n\n

This asks about factors that contribute to heart disease without affecting artery thickness. This is somewhat relevant but doesn’t directly address the key issue of whether pollution is actually causing the observed artery thickening. It’s more about alternative pathways to heart disease rather than alternative explanations for the artery findings.<\/p>\n\n\n\n

D. any of the volunteers whose arteries were among the thickest at the end of the study had arteries that were among the thickest when the study began<\/p>\n\n\n\n

This asks whether people with the thickest arteries at the end also started with thick arteries. This addresses whether the study measured actual change over time versus pre-existing differences, which is important for the study design but doesn’t directly help evaluate the causal claim about pollution.<\/p>\n\n\n\n

E. any other environmental factors tend, in cities with significant amounts of air pollution, to act to thicken arterial walls more quickly than they do in cities with lower amounts of air pollution<\/p>\n\n\n\n

This directly targets the main logical gap by asking whether other environmental factors in polluted cities might be causing the artery thickening instead of pollution itself. If other factors are responsible, this would significantly weaken the conclusion. If pollution is the only relevant factor, this would strengthen the conclusion. This information is most helpful for assessing the force of the evidence.<\/p>\n","protected":false},"excerpt":{"rendered":"

In a study, researchers repeatedly measured the thickness of a specific artery in each of thousands of volunteers over several years. The researchers found during the study that the artery became thicker more quickly in individuals who lived in cities with significant air pollution. 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